Literraly speaking - - Brain Jan 23, 2018 7:25:42 GMT -7
Post by JustJohn or JJ on Jan 23, 2018 7:25:42 GMT -7
Mad Cow disease: Is it really Manganese Madness?
The Idaho Observer has taken a dissident position on every
disease scare that exists. In each case, whether its AIDS,
SARS, West Nile, small/monkey/cow/mousepox, anthrax, the
“official” stories used to scare people do not
stand up to scientific scrutiny, logic or even field
Mad cow is no different except in one
significant way: Prion diseases are currently all up and
down the food chain and all creatures with brains have what
it takes to be ravaged by mutated prions. What on earth has
caused this global propensity for madness? The answer to
that is why people must believe the official lies about mad
Mad Cow or Manganese Madness?
From Friday's Journal
Mad Cow disease has been big in the news of late and I've
been studying the work of Mark Purdey for some good light
on the subject. Purdey is an English cattle farmer with
personal experience with the disease and an extraordinary
amount of scientific research on the subject under his
Everyone seems now to assume that the cause of mad cow
disease is established and known. It is caused by a
horrible “rogue prion” that jumped from sheep
into cattle when the cattle were fed meat and bone meal
that contained the remains of sheep that had scrapie -- the
sheep version of mad cow disease.
This meat and bone meal is made from the carcasses of all
sorts of animals, road kill, euthanized pets and sick farm
animals -- all ground up and dried and made into powder.
Once lodged in the body of an animal or human, this rogue
prion somehow multiplies itself until it takes over your
brain, fills your brain full of holes like a sponge and
then kills you.
Nothing destroys this rogue prion -- not cooking, not
irradiation, not the passage of time.
Everyone seems now to assume that this has been established
beyond controversy as the true cause of mad cow disease. It
has a strong emotional appeal because most people find the
practice of feeding meat and bone meal to naturally
herbivorous cattle perverse and disgusting.
Many well meaning people are using the concern over mad cow
disease to advocate banning this meat and bone meal as a
feed for cattle and raising cattle and other meat animals
by organic standards. I'm all in favor of organic farming
and I'm all in favor of feeding herbivorous cattle on their
proper herbivorous food. Nevertheless, now that I've
studied Purdey's work, I'm not at all convinced that we've
been given the true story on the real cause of mad cow
disease. Purdey appears to have come closer to the true
cause of mad cow disease than anyone else.
Those who say that the problem of mad cow disease, and its
human equivalent Creutzfeldt-Jacob disease, or CJD, would
disappear if the use of this meat and bone meal as cattle
feed were altogether banned, while well-intentioned and
advocating something that I certainly agree should be done,
may not be right.
For instance, Purdey notes that the meat and bone meal was
banned in Britain in 1988, but nevertheless 40,000 cattle
born after the ban have come down with mad cow disease.
Further, he notes that in Ireland, Portugal, and France,
there have been more cases of mad cow disease reported in
cattle born AFTER the meat and bone meal feed was banned in
these countries, than were being reported before the bans.
So the real cause of mad cow disease may very well lie
elsewhere than what we have all been told.
Phosmet and the warble fly
Mark Purdey's interest in mad cow disease began in 1982
when the English government demanded that all cattle be
treated with large doses of an organophosphate insecticide
called Phosmet, applied along their spines in a systemic
formulation designed to go through the animal's skin and
make its system toxic to the warble fly.
Purdey was an organic farmer and didn't want to do it. He
took his case to court where he prevailed in 1984. The next
year, mad cow disease appeared in Britain and Purdey
immediately suspected it was connected to this warble fly
eradication program. He noticed that no cows born and
raised on organic farms and not treated with Phosmet, ever
developed mad cow disease, although the organic standards
at the time allowed feeding these animals some meat and
bone meal. A few of his cattle developed mad cow disease
but they were all animals he had purchased and brought in
from conventional farms.
Purdey thought that the advocates of the “rogue prion
in the meat-and-bone-meal” theory hadn't done a very
good job of explaining how this rogue prion was created in
the first place. Purdey also found there were no
explanations as to how these rogue prions replicated
themselves once they had invaded their host. Therefore,
Purdey reasoned, the meat-and-bone-meal theory cannot
explain how the rogue prions could eventually gain the
force of numbers to turn one's brain into a sponge.
This “rogue” prion is not a virus, it is not a
bacteria, it is not a fungus, it is not a tiny parasitic
animalcule. It is not a living organism of any sort. It is
a protein molecule. In high school biology class we learn
that protein molecules do not replicate themselves
sexually; we learn that proteins are assembled upon demand
from resources found in our bodies and manufactured
according to codes and templates for their structure
contained in our DNA.
Thus far I have not heard any of the advocates of the
popular theory explain this unique self-multiplying
property of this particular prion protein molecule. To my
knowledge, no other protein molecule in the universe has
ever demonstrated such behavior.
The Mad Cowboy
I heard Howard Lyman, the Mad Cowboy, in a radio interview
recently. Lyman, author of a book entitled, “The Mad
Cowboy,” popularized the subject of mad cow disease in
America with his appearance on the Oprah Winfrey show in
1996. I liked listening to Lyman as he's obviously a very
well-intentioned man who has dedicated his life to the
promotion of organic farming. I couldn't help but notice,
however, that his presentation did not contain any science
to back up his explanation of the cause and spread of mad
The scientific cowboy
Purdey, on the other hand, has been proceeding logically to
determine how the prions replicate and how mad cow really
spreads. Suspecting Phosmet was involved but not wanting to
jump to any conclusions, Purdey hired Dr. Stephen Whatley
of the Institute of Psychiatry in London, to subject brain
cell cultures to doses of Phosmet to see if this would
create the rogue prion. As I've come to understand, prion
proteins are normal constituents of nerve and brain tissue
but deformed, abnormal prions are being found in the brains
of the poor wretched creatures who die from mad cow
Whatley's tests caused some deformation of the prion
protein, but not in the precise configuration found in the
mad cow disease. So Purdey went back to square one and
continued researching further, with a willingness to
abandon his initial hypothesis if the evidence could not be
found to support it.
Purdey observed that the mad cow disease, and its human
form CJD, and the same disease found amongst deer and
referred to as chronic wasting disease (CWO), were
occurring in clusters in certain areas here and there
around the world.
This was another point where he found the popular theory
not making sense. If mad cow disease were really caused by
the cattle eating meat and bone meal, and if CJD were
really caused by people eating beef, then the disease
should be occurring at random over broad areas, not in
clusters in, certain small limited areas.
Purdey also observed that Britain was exporting large
amounts of meat and bone meal to a number of countries such
as India, South Africa and Saudi Arabia, and yet the cattle
there were not developing mad cow disease.
Further he noted that several antelope in the London Zoo
came down with mad cow disease but they had never eaten
meat and bone meal. He also observed how several cows on an
English government experimental farm, raised totally on
grass and silage with no meat and bone meal, developed mad
These things just didn't add up to support the official
theory. Since the disease was occurring in clusters in
certain limited areas, Purdey logically suspected that
environmental factors -- something peculiar to the
environment of these areas -- was likely the cause of prion
(I recently heard a radio news report this morning about a
cluster of CJD cases appearing in a town in New Jersey;
more confirmation of Purdey's observations).
Common environmental factors discovered
Purdey traveled to as many mad cow disease cluster areas --
or maybe I should call them spongiform encephelopathy
cluster areas, since they included CJD and CWD areas as
“Spongiform” means that it's full of little holes
like a sponge; “encephelo” means brain, and
“pathy” means disease. So “spongiform
encephelopathy” is just a fancy name for a disease
that fills your brain full of little holes so it looks like
Anyway, Purdey traveled to as many areas around the world
as he could get to, where clusters of the spongiform
encephelopathy diseases were occurring, to examine the
local environments. Purdey was searching for unusual
conditions that may be common among areas reporting
incidence of prion disease.
Purdey reports that, in all these areas, he found a certain
package of similar environmental factors.
One was a very low level of copper in the soil, so that
animals and people living there would tend to be deficient
Another was an abnormally high level of manganese, either
naturally occurring in the soil or, in many cases, as a
result of a local industry polluting the area with
Purdey's investigations revealed is a condition called
“manganese madness” developed with some frequency
by manganese miners, with symptoms essentially the same as
CJD. So it began to look likely that these spongiform
encephelopathy diseases were forms of manganese poisoning.
It is said that CJD is very similar to Alzheimer's disease,
which is generally attributed to metal toxicity from
aluminum and mercury. It makes sense that CJD could be due
to similar metal toxicity from manganese.
Another factor common to mad cow disease clusters, Purdey
found, was either very high levels of ultraviolet light due
to high altitude, or frequent sonic shocks caused by sonic
booms of supersonic aircraft, frequent explosions or, in a
few cases, proximity to a rumbling volcano. So he set out
to discover how this particular package of environmental
factors could be causing brains to turn into sponges.
Scientific support for empirical observations
Purdey then found that his work connected very well with
the work of Dr. David Brown of Cambridge University. Dr.
Brown had demonstrated that normal, healthy prion protein
bonds to copper in the normal, healthy brain and nervous
system. Remember, this prion protein is a normal
constituent of a normal brain; it is abnormal or deformed
prion proteins that are associated with spongiform
Is it the abnormal prion that causes the disease or is it
some unhealthy factor in the environment that causes the
prion to become deformed and abnormal and this leads to the
Purdey published a paper proposing that: Manganese could,
in the presence of copper deficiency in the animal or
human, substitute for copper in the prion protein, thus
producing the abnormal prion found in the brains of
spongiform encephelopathy disease victims.
This was a hypothesis based on his field work in the areas
where clusters of mad cow and CJD were occurring and where,
he observed, all these areas had unusually low levels of
copper in the soil and unusually high levels of manganese
in either the soil or the atmosphere.
Purdey reports that, “David Brown ran the necessary
cell-culture experiments, in which he introduced manganese
into copper-depleted prion protein cell cultures.”
Behold the mutated prion
Amazingly, his experiments produced the key deformation of
the prion protein which the earlier tests using
organophosphate (OP) insecticides like the Phosmet (Purdey
initially suspected as the cause of mad cow disease at the
Institute of Psychiatry) had failed to create. These
experiments represented the first time that malformed prion
protein had been created experimentally as a de novo
So it appears that these experiments definitively answered
the key question of where this abnormal prion came from in
the first place, something I have not heard the advocates
of the conventional theory do with any clarity.
What I have heard them say is that the rogue prion
originally came from sheep with scrapie disease. How the
sheep got it in the first place is not explained. But
because scrapie-diseased sheep brains were included in the
meat and bone meal fed to cattle, the prion got into cattle
where it then proceeded to multiply itself by the
Again they don't explain how the prion multiplies itself.
And if a person eats meat or a beef byproduct such as a
gelatin capsule that contains one of these rogue prions,
again it can multiply itself by the gazillions in the brain
of the unfortunate person until it turns his or her brain
into a sponge and he or she dies a most wretched death.
Supplementation over slaughter
Further, it appears that here we would also have the real
key to preventing spongiform encephelopathy disease -- not
by mass slaughter of healthy animals as the government is
wont to do, on the premise that they may harbor rogue
prions which they will eventually transmit to somebody, and
not by electronically tagging and tracking every animal,
but by simply making sure that the animals -- or we
ourselves -- do not suffer from copper deficiencies in
The conventional theory has made many people worried sick
over getting CJD and they've given up meat eating and
continue to live in anxiety that they may get CJD from a
stray prion that has wormed its devilish way into a gelatin
capsule or bar of soap.
It certainly sounds to me that, if people are concerned
over CJD, the wise thing to do would be to take a mineral
supplement that would assure them of adequate copper. They
might also prevent themselves from getting gray hair that
way, as I've heard Dr. Joel Wallach, the
“dead-doctors-don't-lie” man, say that gray hair
is the result of copper deficiency.
Purdey further reports that researchers at Case Western
University in Cleveland analyzed brain tissue from victims
of CJD. These brain tissues, compared to control brains,
reportedly contained a ten-fold higher level of manganese
and a 50 percent reduced level of copper -- and the
manganese was bonded to the abnormal prions in these CJD
The fanatic vegetarians of course like to use this fear in
the service of their cause but it is wrong to promote
anything on false premises. Let vegetarianism be promoted
by whatever truthful arguments can be mustered up for it,
not by false theories of the cause of spongiform
So it looks like the riddle of what causes these spongiform
encephelopathy diseases is essentially solved. It's a form
of manganese poisoning that can occur in the presence of a
Purdey has some further refinements to add to the picture.
For instance, what about the Phosmet Purdey initially
suspected as the cause of mad cow disease?
Purdey found that Phosmet chelates copper out of the bodies
of the cattle, thus leading to the copper deficiency that
then allowed mad cow disease to develop. There are
organophosphate pesticides, herbicides and fertilizers
humans use for various purposes that can have a similar
effect on us.
More observations in support of the OP/copper/manganese
Purdey says Britain had the highest rate of mad cow disease
because of the compulsory warble fly eradication program
that required all farmers to treat their cattle with high
doses of systemic Phosmet applied along the spine. Other
countries that experienced some outbreak of the disease but
not as much as Britain either did not have such a
compulsory program or used the organophosphates in lower
doses or non-systemically.
Another product Purdey implicates in mad cow disease is
artificial milk substitute often fed to calves, which have
up to 1,000 times the level of manganese found in natural
Purdey observed that the mad cow, CJD and CWD cluster areas
all were subject either to frequent sonic shocks or very
high levels of ultraviolet light.
Copper is used in wires as an efficient conductor of
electricity; manganese is used in batteries to store
electricity. Purdey hypothesizes that the normal prion
protein, bonded to copper, facilitates a smooth flow of the
energy brought into the nervous system via sound and light.
Conversely, when manganese substitutes for copper, energy
would have a tendency to be stored rather than allowed to
flow. This could result in explosive releases of energy,
thereby progressively deteriorating the brain tissues in
the spongiform pattern.
It's quite an ingenious explanation. I haven't heard the
proponents of the official theory advance a scientifically
viable explanation as to why the rogue prion causes
spongiform deterioration of the brain.
Purdey also suspects the sonic shocks from supersonic
airplanes could be altering the magnetic qualities of
manganese in such a way as to promote an even more rapid
degeneration of the brain tissues.
The kuru copper connection
Another case that is frequently mentioned in discussions of
mad cow disease, is the disease called “kuru”
that has afflicted many members of the Fore tribe in New
Being essentially the same as CJD, kuru is always
attributed to traditions of cannibalism specific to these
The public readily accepts this explanation since
cannibalism is morally and emotionally repugnant to most
people and, therefore, it seems a sort of divine or poetic
justice that it would lead to a horrible disease like kuru.
It's sort of similar to the way sodomy is repugnant to
people of normal sexual desires, and so some see it as a
sort of divine or poetic justice that it's practice be
blamed for transmitting AIDS.
However, Purdey observed that many other New Guinea tribes
were cannibals but only the Fore were getting kuru.
Curiously, the Fore people had been cannibals for centuries
but had only started getting kuru after World War II.
Purdey traveled there to examine the environment, and found
the local soils deficient in copper.
Manganese/aluminum alloy and kuru-type madness
Completing the picture, during World War II some Japanese
airplanes had crashed in the area. The Fore people had made
utensils and cooking pots from the manganese-aluminum metal
they salvaged from these airplanes -- a likely source of
Purdey also found that a cluster of CJD cases had developed
in the Fuji River valley in Japan, downwind from the
factory where the manganese-aluminum panels for these
airplanes were manufactured, where the atmosphere was
tainted with manganese-laden emissions from the factory
Dissidence deadlier than disease
Science these days is inevitably entangled with politics,
and Purdey found that in advancing his perspective on the
true cause of mad cow disease, he evidently displeased some
powerful political interests. His house burned down. His
lawyer, who had helped him with his court case against the
government's mandatory Phosmet program and his
veterinarian, who was assisting in his research, both died
in strange auto accidents. Attempts were made to slander
and discredit him morally and professionally.
What political interests would want to interfere with his
efforts to bring some objective science to the question of
the cause of mad cow disease?
The chemical industry that makes $billions from
organophosphate sales would well have an interest in not
having these chemicals linked to causing outbreaks of prion
Slaughter the animals, kill the family farm
Purdey also sees an interest of corporate farming here:
Massive, multinational corporations such as Arthur Daniels
Midland are putting vast acreages of farmland worldwide
under cultivation. They are growing genetically engineered
grains, vegetables, fruits and legumes heavily treated with
chemical pesticides and chemical fertilizers.
Their competition is traditional family farmers who raise
crops and livestock without chemicals. If word got out that
global madness through mutated prions was linked to
genetically engineered crops treated with OPs,
multinational corporate agribusiness would suffer a public
relations disaster from which it may never recover.
It would well suit these agribusiness interests to put
traditional family farmers out of business. Since there is
no science to support the mass slaughter of infected
animals, it is logical to suspect that public fear of
eating contaminated meat or is nurtured in the press to
justify government slaughter programs. Rather than
exterminating the rogue prions, government slaughter
programs are helping to exterminate agribusiness'
competition: The family farmer.
Big Brother implications
The official theory also furnishes a convenient rationale
to mandate electronic tagging and tracking of all livestock
via microchips. Developing the livestock surveillance
infrastructure lays the groundwork for applying such a
surveillance system to people.
Chemical/copper connection known for half a century
In an article entitled “The Grammar of BSE
Panic” in the February, 2004 edition of “Acres
U.S.A.” we find these words:
“Writing some 50 years ago in SOIL, GRASS &
CANCER” Andre Voisin noted how certain chemicals
interfered with the uptake of copper even when that
nutrient was available in the pastures.
“He called this 'disease' epizootic ataxia. It sounds
like bovine spongiform encephelopathy to us, and the
university-blessed grinding of cats, dogs, cows, chickens
and horses to make cattle-cake was not even a blip on the
radar screen at that time...After England experienced BSE
cases in animals, now identified by prions bent out of
shape, the defenders of toxic-genetic chemicals postulated
an infectious disease agent of some sort and resultant
“The answer was to shoot herds and to construct a
theory with a politically-correct etiology. The only rule
seemed to be, 'Do not implicate phosphate factories spewing
their pollution into the ambient air, and never, never
indict Phosmet poured on animal spines...There are several
chemical agents quite capable of turning a brain into a
sponge. Mercury and Minimata disease come to mind.”
Official story does not pass basic scientific muster
In the same publication indicated above, a letter from the
Minnesota COACT organization( www.coact.org ) said,
“BSE fails to fulfill what is known as 'Koch's
postulates' -- the yardstick for gauging whether a given
disease stems from infectious origins. Briefly, they are
1. The causal agent is found in all symptomatic animals;
2. The causal agent is not found with other diseases; and
3. The causal agent can be isolated and cultured, then
reproduced in a susceptible host.”
COACT also noted that, “A BSE prion is a protein
particle that does not contain DNA or RNA.”
So without any DNA or RNA how does one of these bloody
things jump off your hamburger and into your body and so it
can madly replicate itself until your brain has been turned
into a sponge?
Since the powers-that-be have an interest in maintaining
the rogue prion theory as the cause of mad cow disease in
public perception, you won't hear of Purdey's work being
reported in the dominant media.
But what Purdey says makes sense and his theories are
supported by 50 years of published science where the
official theory would not pass a high school biology test.
So I've put on my journalist's hat and have written this to
do my bit to get the word out to the public, because that's
the only way we can hope to get the truth out.
Thomas Jefferson said, “If the people lead, then
eventually the leaders will follow.”
If we can make the truth about mad cow disease common
enough knowledge amongst the general public, then the
authorities of the scientific world will finally accept it.
Unfortunately though, if that happens, the authorities of
the scientific world will not give Purdey the credit he
deserves but will claim to have discovered it themselves:
“Can't allow the peasants to think they can do
It's like 75 years ago the health nuts were saying you
could prevent cancer by eating a lot of fresh fruits and
vegetables. The medical authorities debunked it and said
your diet had nothing to do with whether or not you got
cancer. But the health nuts kept on, and eventually the
general public came to understand the relationship between
diet and cancer that the medical authorities have suddenly
discovered you can reduce your risk of cancer by eating a
lot of fresh fruits and vegetables.
But they cite their own studies in support of this radical
concept and do not give the health nuts of 75 years ago any
credit for having known it all along.
One problem with communicating Purdey's perspective is that
it takes a little bit of study to understand it. The
official theory is very easy to understand and emotionally
grabbing. “They're feeding rotten meat to cows that
are supposed to eat grass!” Which I agree is not the
right thing to do, but that doesn't prove it's really the
cause of mad cow disease.
Purdey's writing by contrast is fairly technical and I had
to read it through about six times before I understood it.
Not many people will bother to do that. Although, since I'm
just a high school dropout and not very bright, most people
wouldn't have to study it near as much as I did to
understand it. So I've written this to try to put Purdey's
perspective in as plain and simple language as I can to try
to help more people understand it.
So, if you think this article is saying something true and
important, HELP GET THE WORD OUT! Make more copies of this,
put it up on websites, email it or snail mail it to as many
people as you can. And, of course, it would be great if
you'd read Purdey's work for yourself and don't depend just
on me for your understanding of it.
The difference between us and the dominant media is that we
do the best we can to tell each other the truth so we can
make the most of our lives and raise our children in a
decent world whereas the dominant media engages
psychological warfare against you, in order to deceive and
mislead you into supporting its big business advertisers. (DWH)
You can read Purdey's work for yourself at his website, which is at www.markpurdey.com .